Evidence for God from Science

In five different posts in this thread I have argued that in an old species as E coli and with a stable environment there will be no (or very few) beneficial mutations. You have never commented that statement! Instead you have continued to say things as: Post #49 I personally wouldn't expect to see ...

In my previous post I neglected to address this issue... The malaria examples demonstrate that coordinated mutations in the real world are not necessarily a function of beneficial mutations. If coordinated mutations were typically a function beneficial mutations, then we would expect to see 100 mil...

Especially when the overwhelming majority of observed beneficial mutations degrade or remove genetic information instead of altering (like the malaria examples) or even more infrequently adding genetic information. Beneficial mutations are indeed very uncommon. Especially when the environment isn’t...

Especially when the overwhelming majority of observed beneficial mutations degrade or remove genetic information instead of altering (like the malaria examples) or even more infrequently adding genetic information. Beneficial mutations are indeed very uncommon. Especially when the environment isn’t...

We have a real life example on the table of two mutations working together to form a new function in malaria's resistance to chloroquine. Due to the exponential nature of the two mutations occurring, you presume that this particular example involves two non-beneficial mutations working together to ...

But to spell it out explicitly... Nills said... But first, there is an enormous amount of evidence of multiple beneficial mutations (which by definition give new functions). So the question is whether a lot of these small increments of functions can produce complex functions. There is an enormous a...

Summarising the discussion so far it seems that you now understand that several beneficial mutations can give new functions faster than several non-beneficial mutations. Now, it seems that you doubt that many beneficial mutations can produce complex functions. Oh I understand the premise that sever...

But Malaria is not alone. There are many diseases out there in the world, and scientists are constantly developing new drugs to fight these diseases. If multiple beneficial mutations are able to create new functions exponentially faster than multiple non-beneficial mutations (like malaria developin...

Getting back to the malaria example Since the observed difference between malaria developing resistance to atovaquone and chloroquine is exponential, then using your logic, that would indicate that malaria's resistance to chloroquine involved 'non-beneficial' mutations of some sort. Would you agree...

I will now again try to make my argument clearer, showing which assumptions I make, without using any new terminology. My presumptions are: - Mutations occur randomly and the probability of one specific mutation to occur in some specific DNA point during the lifetime of one individual is a fixed va...

I understand your premise, but I don't see any empirical evidence to support your premise. Which premise are you talking about? Your premise presumes that sequential mutations will produce a new function involving coordinated mutations at a faster rate than coordinated simultaneous mutations. Now y...

Natural evolution according to the evolution theory is about evolution using mutations AND selection, mostly by creating mutations that are advantageous by themselves and then selected for. Agreed The malaria case is about how two mutations that aren’t beneficial alone cooperate in some rare cases ...

Natural evolution according to the evolution theory is about evolution using mutations AND selection, mostly by creating mutations that are advantageous by themselves and then selected for. The malaria case is about how two mutations that aren’t beneficial alone cooperate in some rare cases to creat...

You say (numbering inserted): “So my argument is based on 1. observed empirical evidence from nature (the exponential nature of coordinated mutations in malaria), 2. empirical evidence in the lab (observed unguided evolution degrades and loses information), and 3. an extrapolation of the observed b...

“So the observed behavior of random mutation in the real world demonstrates that random mutation is incapable of producing the either the scope or rate of change that we see in either the fossil record or the DNA of life today. That's what the observed behavior of random mutation in the real world ...